Outside of its pain-relieving effects, conolidine has shown a favorable safety margin in preclinical toxicology assessments. Contrary to opioids, which regularly induce respiratory depression at better doses, conolidine has not manufactured signs of important respiratory suppression. Also, behavioral assessments in murine styles propose an absence
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We shown that, in contrast to classical opioid receptors, ACKR3 will not induce classical G protein signaling and is not modulated through the classical prescription or analgesic opioids, including morphine, fentanyl, or buprenorphine, or by nonselective opioid antagonists including naloxone. In its place, we set up that LIH383, an ACKR3-selective
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